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group_1_presentation_3_-_atrial_fibrillation [2016/04/01 23:41] dheriaj |
group_1_presentation_3_-_atrial_fibrillation [2018/01/25 15:18] (current) |
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- | {{:playground:pic_2.jpg?400x390}} | + | {{:playground:pic_2.jpg?410x390}} |
''Figure 4: The relationship between pressure and time of the atrial \\ contraction, comparing the normal sinus rhythm (left) with the sinus \\ rhythm suffering from atrial fibrillation (right) <sup>[10]</sup>.'' </style> | ''Figure 4: The relationship between pressure and time of the atrial \\ contraction, comparing the normal sinus rhythm (left) with the sinus \\ rhythm suffering from atrial fibrillation (right) <sup>[10]</sup>.'' </style> | ||
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{{:playground:pic_44.jpg?400x400}} | {{:playground:pic_44.jpg?400x400}} | ||
- | http://users.atw.hu/blp6/BLP6/HTML/common/M9780323045827-013-f003.jpg </style> | + | Figure 5: The role of calcium in normal excitation coupling |
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=== Normal Excitation Coupling === | === Normal Excitation Coupling === | ||
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==== Concluding Remarks ==== | ==== Concluding Remarks ==== | ||
- | Continued research in determining the relative roles of ectopic activity, single-circuit reentry and multiple-circuit reentry in maintaining clinical AFib is important to better understand the mechanisms underlying AFib. Answering these questions will lead to new and improved possibilities in prevention and therapy. | + | There are many questions that need to be answered in order to better understand the mechanisms underlying AFib, such as the genes responsible for familial Afib and how they lead to arrhythmia. Continued research in determining the relative roles of ectopic activity, single-circuit reentry and multiple-circuit reentry in maintaining clinical AFib is important to better understand the mechanisms underlying AFib. Answering these questions will lead to new and improved possibilities in prevention and therapy. |
====== References ====== | ====== References ====== |